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The goal of the NIDDK-sponsored Diabetic Complications Consortium (DiaComp) is to advance the study of diabetic complications and promote communication & collaboration between investigators involved in complications research. Toward this goal, DiaComp will:
Sponsor annual meetings in complications-relevant scientific areas; Solicit and fund pilot projects in high impact areas of complications research; Support a website to serve the diabetic complications community.
Perinatal versus adult loss of ULK1 and ULK2 distinctly influences cardiac autophagy and function.
A novel method for visualizing and tracking endogenous mRNA in a specific cell population in pathological neovascularization.
Pathophysiology of Gastroparesis Syndromes Includes Anatomic and Physiologic Abnormalities.
NHE3 in the thick ascending limb is required for sustained but not acute furosemide-induced urinary acidification.
SGLT2 inhibition effect on salt-induced hypertension, RAAS, and Na+ transport in Dahl SS rats.
Progress toward the Clinical Application of Mesenchymal Stromal Cells and Other Disease-Modulating Regenerative Therapies: Examples from the Field of Nephrology.
Botulinum Injection Into the Proximal Intestinal Wall of Diet-Induced Obese Mice Leads to Weight Loss and Improves Glucose and Fat Tolerance.
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Please acknowledge all posters, manuscripts or scientific materials that were generated in part or whole using funds from the Diabetic Complications Consortium(DiaComp) using the following text:
Financial support for this work provided by the NIDDK Diabetic Complications Consortium (RRID:SCR_001415, www.diacomp.org), grants DK076169 and DK115255
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